Grants per year
Personal profile
Research Interests
Ataxias, spinocerebellar ataxia, movement disorders, neurodegenerative diseases, stroke, cerebrovascular disorders.
Certifications and Licenses
| Neurology |
Training Experience
| 1996 | Internship, Saint Francis Hospital, Evanston |
| 1999 | Residency, Univ. of Iowa Hospitals & Clinics |
| 2003 | Postdoctoral Fellowship, Baylor College Of Medicine |
Education/Academic qualification
PhD, Northwestern University Feinberg School of Medicine
… → 1995
MD, University of Poona
… → 1989
Research interests
- Movement Disorders
- Neurogenetics
- Stroke
Fingerprint Dive into the research topics where Puneet Opal is active. These topic labels come from the works of this person. Together they form a unique fingerprint.
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Spinocerebellar Ataxias
Medicine & Life Sciences
Intermediate Filaments
Medicine & Life Sciences
Leucine
Medicine & Life Sciences
Giant Axonal Neuropathy
Medicine & Life Sciences
Nuclear Proteins
Medicine & Life Sciences
Intermediate Filament Proteins
Medicine & Life Sciences
Neurodegenerative Diseases
Medicine & Life Sciences
Ataxia
Medicine & Life Sciences
Network
Recent external collaboration on country level. Dive into details by clicking on the dots.
Grants 2003 2024
Understanding pathophysiological mechanisms underlying neurodegeneration
Goldman, R., Opal, P. & Savas, J. N.
National Institute of Neurological Disorders and Stroke
7/1/19 → 6/30/20
Project: Research project
Giant Axonal Neuropathy
Intermediate Filaments
Pathology
Proteins
Ubiquitin-Protein Ligases
Understanding the Cellular Basis of Movement Disorders
National Institute of Neurological Disorders and Stroke
6/1/19 → 4/30/24
Project: Research project
Spinocerebellar Ataxias
Movement Disorders
Trinucleotide Repeat Expansion
Purkinje Cells
Neurodegenerative Diseases
Spinocerebellar Ataxias
Nanoparticles
Vascular Endothelial Growth Factor A
Angiogenesis Inducing Agents
Neurodegenerative Diseases
Understanding pathophysiological mechanisms underlying Giant Axonal Neuropathy
Muscular Dystrophy Association
2/1/19 → 1/31/22
Project: Research project
Giant Axonal Neuropathy
Intermediate Filaments
Neurites
Cranial Nerve Diseases
Mutation
Research Output 1991 2019
Loss of the dystonia gene Thap1 leads to transcriptional deficits that converge on common pathogenic pathways in dystonic syndromes
Frederick, N. M., Shah, P. V., Didonna, A., Langley, M. R., Kanthasamy, A. G. & Opal, P., Apr 15 2019, In : Human molecular genetics. 28, 8, p. 1343-1356 14 p.Research output: Contribution to journal › Article
Dystonia
Genes
Haploinsufficiency
Nestin
Gliosis
2
Citations
(Scopus)
Self-assembling vascular endothelial growth factor nanoparticles improve function in spinocerebellar ataxia type 1
Hu, Y. S., Do, J., Edamakanti, C. R., Kini, A. R., Martina, M., Stupp, S. & Opal, P., Feb 1 2019, In : Brain. 142, 2, p. 312-321 10 p.Research output: Contribution to journal › Article
Spinocerebellar Ataxias
Nanoparticles
Vascular Endothelial Growth Factor A
Nerve Growth Factors
Neurodegenerative Diseases
1
Citation
(Scopus)
The role of neurofilament aggregation in neurodegeneration: Lessons from rare inherited neurological disorders
Didonna, A. & Opal, P., May 16 2019, In : Molecular neurodegeneration. 14, 1, 19.Research output: Contribution to journal › Review article
Open Access
Intermediate Filaments
Nervous System Diseases
Intermediate Filament Proteins
Transport Vesicles
Neurotransmitter Receptor
4
Citations
(Scopus)
ANP32A regulates histone H3 acetylation and promotes leukemogenesis
Yang, X., Lu, B., Sun, X., Han, C., Fu, C., Xu, K., Wang, M., Li, D., Chen, Z., Opal, P., Wen, Q., Crispino, J. D., Wang, Q. F. & Huang, Z., Jul 1 2018, In : Leukemia. 32, 7, p. 1587-1597 11 p.Research output: Contribution to journal › Article
Phosphoproteins
Acetylation
Leucine
Histones
Histone Code
8
Citations
(Scopus)
Mutant ataxin1 disrupts cerebellar development in spinocerebellar ataxia type 1
Edamakanti, C. R., Do, J., Didonna, A., Martina, M. & Opal, P., Jun 1 2018, In : Journal of Clinical Investigation. 128, 6, p. 2252-2265 14 p.Research output: Contribution to journal › Article
Spinocerebellar Ataxias
Purkinje Cells
Interneurons
Stem Cells
Cerebellar Diseases