A Functional Genomic Fingerprint of Chronic Stress in Humans: Blunted Glucocorticoid and Increased NF-κB Signaling

Keyphrases

• Caregivers 100%
• Monocytes 100%
• Response Element 100%
• NF-B 100%
• Control Subjects 100%
• Genomic Fingerprints 100%
• Functional Genomics 100%
• Chronic Stress 100%
• Cancer Patients 50%
• Family Caregivers 50%
• Inflammatory Response 50%
• Vulnerability 50%
• Brain Cancer 50%
• Inflammatory Markers 50%
• C-reactive Protein 50%
• Normal Activity 50%
• Transcriptional Control 50%
• Inflammatory Mechanisms 50%
• Salivary Cortisol 50%
• Medical Illness 50%
• Diminished Expression 50%
• Stress-related 50%
• Peripheral Blood Monocytes 50%
• Chronic Stressors 50%
• Interleukin-1 Receptor Antagonist (IL-1Ra) 50%
• Cortisol Secretion 50%
• Expression Microarray 50%
• Adrenocortical 50%
• Transcription Control 50%
• Relative Height 50%
• Bearing Response 50%
• Inflammatory Transcription Factors 50%
• Functional Resistance 50%

Immunology and Microbiology

• Finger Dermatoglyphics 100%
• Chronic Stress 100%
• Glucocorticoid 100%
• DNA Responsive Element 66%
• Inflammation Response 33%
• Interleukin 1 Receptor Antagonist 33%
• Hydrocortisone Release 33%
• Transcription Factors 33%
• C-Reactive Protein 33%
• Cortisol 33%

Neuroscience

• Glucocorticoid 100%
• Functional Genomics 100%
• Cortisol 66%
• Response Element 66%
• Interleukin 1 Receptor Antagonist 33%
• Transcription Factors 33%
• C-Reactive Protein 33%
• Brain Cancer 33%
• Synapsin I 33%
• Microarrays 33%