Ablation of intact hypothalamic and/or hindbrain TrkB signaling leads to perturbations in energy balance

Keyphrases

• Ablation 100%
• Energy Balance 100%
• Hindbrain 100%
• Tropomyosin Receptor Kinase B 100%
• NTRK2 80%
• Brain-derived Neurotrophic Factor 40%
• Nkx2.1 40%
• PHOX2B 40%
• High-fat Diet 30%
• Adiposity 20%
• Brain Regions 20%
• Glucose Homeostasis 20%
• Metabolic Control 20%
• Food Intake 20%
• Hypothalamus 20%
• Heterozygous Mice 20%
• Increased Body Weight 20%
• Central Regulation 20%
• Chow Diet 20%
• Hyperphagia 20%
• Weight Difference 20%
• B Deficiency 20%
• Body Adiposity 20%
• Metabolic Phenotype 10%
• Metabolic Parameters 10%
• Energy Expenditure 10%
• Core Body Temperature 10%
• Motor Neuron 10%
• Locomotor Activity 10%
• Blood Glucose 10%
• Cardiovascular Function 10%
• Female Mice 10%
• Respiratory Function 10%
• Littermate Controls 10%
• Genetic Evidence 10%
• Serum Leptin 10%
• Human Obesity 10%
• Serum Insulin Levels 10%
• Fed Condition 10%
• Endogenous Role 10%
• Digestive Function 10%
• Body Weight Homeostasis 10%
• High-fat Chow 10%

Neuroscience

• Kinase 100%
• Hindbrain 100%
• Energy Balance 100%
• Tropomyosin 100%
• Receptor 100%
• Brain-Derived Neurotrophic Factor 36%
• Hypothalamus 18%
• Polyphagia 18%
• Leptin 9%
• Locomotor Activity 9%
• Energy Expenditure 9%
• Motor Neuron 9%

Biochemistry, Genetics and Molecular Biology

• Energy Balance 100%
• Tropomyosin Receptor Kinase B 100%
• Body Weight 60%
• Brain-Derived Neurotrophic Factor 40%
• Glucose Homeostasis 20%
• Metabolic Regulation 20%
• Wild Type 10%
• Respiratory Function 10%
• Cardiovascular Function 10%
• Genetics 10%
• Glucose Blood Level 10%
• Locomotion 10%
• Motor Neuron 10%
• Insulin Blood Level 10%
• Core Temperature 10%
• Energy Expenditure 10%
• Digestive Function 10%
• Leptin 10%