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Animal Models of Spinocerebellar Ataxia Type 1
Puneet Opal
*
, Harry T. Orr
*
Corresponding author for this work
Neurology
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Keyphrases
Spinocerebellar Ataxia
100%
Ataxin-1
100%
Inclusion Formation
25%
Nuclear Inclusions
25%
Phosphorylation
12%
Neurodegenerative Diseases
12%
Disease Progression
12%
Nuclear Localization
12%
Toxic Effects
12%
Transcriptional Regulator
12%
Neurodegeneration
12%
Disease-modifying Therapy
12%
RNA Processing
12%
Hypothesis-driven
12%
Mutant Protein
12%
Purkinje Cell
12%
Processing Route
12%
Protein Clearance
12%
Encoded Protein
12%
Protein Folding
12%
Polyglutamine Repeat
12%
Protein Processing
12%
Single Amino Acid
12%
Pathogenic Potential
12%
Neuroscience
Ataxin 1
100%
Spinocerebellar Ataxia
100%
Serine
25%
Protein Folding
12%
Purkinje Cell
12%
Neurodegenerative Disorder
12%
Amino Acid
12%
RNA Processing
12%
Polyglutamine
12%
Mutant Protein
12%
Alanine
12%
Neurodegeneration
12%
Posttranslational Modification
12%
Biochemistry, Genetics and Molecular Biology
Animal Models
100%
Ataxin 1
100%
Serine
25%
Wild Type
12%
Posttranslational Modification
12%
Amino Acids
12%
RNA Processing
12%
Protein Folding
12%
Purkinje Cell
12%
Mutant Protein
12%
Alanine
12%