Cdk5 protein inhibition and Aβ42 increase BACE1 protein level in primary neurons by a post-transcriptional mechanism: Implications of CDK5 as a therapeutic target for Alzheimer disease

Katherine R. Sadleir, Robert Vassar*

*Corresponding author for this work

Research output: Contribution to journal › Article › peer-review

51 Scopus citations

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Medicine and Dentistry

Neuroscience

Biochemistry, Genetics and Molecular Biology