Comparison of myeloid neoplasms with nonclassic 3q26.2/MECOM versus classic inv(3)/t(3;3) rearrangements reveals diverse clinicopathologic features, genetic profiles, and molecular mechanisms of MECOM activation

Juehua Gao, Sandeep Gurbuxani, Taylor Zak, Masha Kocherginsky, Peng Ji, Firas Wehbe, Qing Chen, Yi Hua Chen, Xinyan Lu, Lawrence Jennings, Olga Frankfurt, Jessica Altman, Madina Sukhanova*

*Corresponding author for this work

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