Correction to: Activation of PKCδ and p38δ MAPK during okadaic acid dependent keratinocyte apoptosis (Archives of Dermatological Research, (2007), 299, 2, (71-83), 10.1007/s00403-006-0727-4)

In this article, the beta-actin bands in Figure 3C were incorrect. The correct beta-actin bands from original data are now included in Fig. 3C; the figure should have appeared as shown below. (Figure presented.) OA treatment activates keratinocyte apoptotic responses. a Keratinocytes, grown on coverslips, were treated with or without 100 nM OA. At 24 h post-infection the cells were stained with MitoSensor reagent and dye uptake was examined by fluorescence microscopy. The arrows indicate the transparent spherical structures. b Keratinocytes were treated with or without 100 nM OA for 24 h. The cells were then fractionated and cytochrome c level, in cytosol and mitochondrial fractions, was measured by immunoblot. Successful separation of the mitochondrial and cytosolic fractions was confirmed by detection of COX4 (a mitochondrial marker) and β-actin (a cytosol marker). c OA treatment results in procaspase-3 and PARP cleavage. Keratinocytes were treated for 48 h in the presence of absence of 100 nM OA and total cell extracts were fractionated on a 10% gel for immunoblot with anti-caspase 3, anti-PARP and anti-β-actin.