Distinct pathophysiological mechanisms of cardiomyopathy in hearts lacking dystrophin or the sarcoglycan complex

Keyphrases

• Dystrophin 100%
• Cardiomyopathy 100%
• Pathophysiological Mechanisms 100%
• Sarcoglycan 100%
• Limb-girdle muscular Dystrophy 30%
• Myocytes 30%
• Skeletal muscle 20%
• Duchenne muscular Dystrophy 20%
• Mouse Model 10%
• Fibrosis 10%
• Cardiac Function 10%
• Tight 10%
• Hemodynamic Assessment 10%
• Disease Mechanisms 10%
• Membrane Binding 10%
• Unique Mechanism 10%
• Contracture 10%
• Gene-deficient Mice 10%
• Reduced Survival 10%
• Induced Damage 10%
• Cytoskeletal Proteins 10%
• Catheter-related 10%
• Poor Function 10%
• Dobutamine 10%
• Dobutamine Stress 10%
• Normal Compliance 10%
• Mechanical Damage 10%
• Passive Extension 10%
• Dystrophic Cardiomyopathy 10%
• Dystrophic Mice 10%
• Passive Compliance 10%

Biochemistry, Genetics and Molecular Biology

• Dystrophin 100%
• Sarcoglycan 100%
• Skeletal Muscle 20%
• Cardiac Muscle Cell 20%
• Dobutamine 20%
• Mouse Model 10%
• Hemodynamic 10%
• Cytoskeletal Protein 10%
• Heart Function 10%