Endoplasmic Reticulum Stress Regulates Hepatic Bile Acid Metabolism in Mice

Keyphrases

• Endoplasmic Reticulum Stress (ER stress) 100%
• Bile Acid Metabolism 100%
• Bile Acids 40%
• Hepatocytes 20%
• Mouse Cells 20%
• Hepatic Expression 20%
• HepG2 Cells 20%
• Primary Bile Acid 20%
• Bile Acid Transport 20%
• Gene Expression 10%
• Liver 10%
• Bile Salts 10%
• Inflammatory Cytokines 10%
• Stress Response 10%
• C57BL 10%
• Plasma Parameters 10%
• Cholesterol 10%
• Novel Functions 10%
• Regulatory Pathways 10%
• Endoplasmic Reticulum 10%
• Acid Removal 10%
• Cholestasis 10%
• Fgf15 10%
• Hydroxylase 10%
• Cytokine Activation 10%
• Acid Content 10%
• Synthetic Pathway 10%
• CYP7A1 10%
• Efflux Pump 10%
• Bile Salt Export Pump 10%
• Synthetic Enzyme 10%
• Intestinal Fibroblasts 10%
• Adenosine Triphosphate-binding Cassette 10%
• HepG2 Cancer Cells 10%
• Small Heterodimer Partner 10%
• Mouse Hepatoma 10%

Biochemistry, Genetics and Molecular Biology

• Endoplasmic Reticulum Stress 100%
• Bile Acid Metabolism 100%
• Bile Acid 60%
• Bile Acid Synthesis 50%
• Hepatocyte 20%
• Inflammatory Cytokine 10%
• Enzyme 10%
• Blood Level 10%
• C57BL 6 Mouse 10%
• Adenosine Triphosphate 10%
• Oxygenase 10%
• Fibroblast Growth Factor 10%
• Blood Plasma 10%