H2-M3-restricted T cell response to infection

Angela Colmone; Chyung Ru Wang

doi:10.1016/j.micinf.2006.03.020

H2-M3-restricted T cell response to infection

Angela Colmone, Chyung Ru Wang^*

^*Corresponding author for this work

Microbiology-Immunology

Research output: Contribution to journal › Short survey › peer-review

20 Scopus citations

Abstract

H2-M3 is a major histocompatibility complex class Ib molecule that presents N-formylated peptides to specific CD8⁺T cells. Prokaryotic, but not eukaryotic, translation begins with the addition of N-formyl methionine, suggesting a role for these H2-M3-restricted T cells in response to bacterial infection. Indeed, these cells constitute a non-redundant "early" component of anti-microbial response.

Original language	English (US)
Pages (from-to)	2277-2283
Number of pages	7
Journal	Microbes and Infection
Volume	8
Issue number	8
DOIs	https://doi.org/10.1016/j.micinf.2006.03.020
State	Published - Jul 2006

Keywords

Listeria
Major histocompatability complex
T-Lymphocytes

ASJC Scopus subject areas

Microbiology
Immunology
Infectious Diseases

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

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10.1016/j.micinf.2006.03.020

Cite this

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title = "H2-M3-restricted T cell response to infection",

abstract = "H2-M3 is a major histocompatibility complex class Ib molecule that presents N-formylated peptides to specific CD8+T cells. Prokaryotic, but not eukaryotic, translation begins with the addition of N-formyl methionine, suggesting a role for these H2-M3-restricted T cells in response to bacterial infection. Indeed, these cells constitute a non-redundant {"}early{"} component of anti-microbial response.",

keywords = "Listeria, Major histocompatability complex, T-Lymphocytes",

author = "Angela Colmone and Wang, {Chyung Ru}",

note = "Funding Information: We thank Honglin Xu and Michael Zimmer for critical reading of this manuscript. This work was supported by National Institutes of Health grant AI40310 (to C.-R. W.). Copyright: Copyright 2011 Elsevier B.V., All rights reserved.",

year = "2006",

month = jul,

doi = "10.1016/j.micinf.2006.03.020",

language = "English (US)",

volume = "8",

pages = "2277--2283",

journal = "Microbes and Infection",

issn = "1286-4579",

publisher = "Elsevier Masson SAS",

number = "8",

}

TY - JOUR

T1 - H2-M3-restricted T cell response to infection

AU - Colmone, Angela

AU - Wang, Chyung Ru

N1 - Funding Information: We thank Honglin Xu and Michael Zimmer for critical reading of this manuscript. This work was supported by National Institutes of Health grant AI40310 (to C.-R. W.). Copyright: Copyright 2011 Elsevier B.V., All rights reserved.

PY - 2006/7

Y1 - 2006/7

N2 - H2-M3 is a major histocompatibility complex class Ib molecule that presents N-formylated peptides to specific CD8+T cells. Prokaryotic, but not eukaryotic, translation begins with the addition of N-formyl methionine, suggesting a role for these H2-M3-restricted T cells in response to bacterial infection. Indeed, these cells constitute a non-redundant "early" component of anti-microbial response.

AB - H2-M3 is a major histocompatibility complex class Ib molecule that presents N-formylated peptides to specific CD8+T cells. Prokaryotic, but not eukaryotic, translation begins with the addition of N-formyl methionine, suggesting a role for these H2-M3-restricted T cells in response to bacterial infection. Indeed, these cells constitute a non-redundant "early" component of anti-microbial response.

KW - Listeria

KW - Major histocompatability complex

KW - T-Lymphocytes

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U2 - 10.1016/j.micinf.2006.03.020

DO - 10.1016/j.micinf.2006.03.020

M3 - Short survey

C2 - 16824777

AN - SCOPUS:33748184090

VL - 8

SP - 2277

EP - 2283

JO - Microbes and Infection

JF - Microbes and Infection

SN - 1286-4579

IS - 8

ER -

H2-M3-restricted T cell response to infection

Abstract

Keywords

ASJC Scopus subject areas

UN SDGs

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