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Human sodium channel myotonia: slowed channel inactivation due to substitutions for a glycine within the III‐IV linker.
H. Lerche*, R. Heine, U. Pika, A. L. George, N. Mitrovic, M. Browatzki, T. Weiss, M. Rivet‐Bastide, C. Franke, M. Lomonaco
*Corresponding author for this work
Research output: Contribution to journal › Article › peer-review
183
Scopus
citations