Interstitial cystitis: Animal models

Tony Buffington; Michael R. Ruggieri; David J. Klumpp

doi:10.1007/978-3-319-61449-6_6

Interstitial cystitis: Animal models

Tony Buffington^*, Michael R. Ruggieri, David J. Klumpp

^*Corresponding author for this work

Urology

Research output: Chapter in Book/Report/Conference proceeding › Chapter

3 Scopus citations

Abstract

In 1990, although the etiology and pathogenesis of the condition then referred to as “interstitial cystitis” (IC) was admitted to be largely or even completely unknown, the widely held implicit working assumptions held that it is a bladder disease. These assumptions implied that if we could discover and correct what was wrong with the bladder itself then we could cure the condition. These assumptions have proved to be largely off base. At that time, the previous suggestions that IC might have a primarily autoimmune or infectious etiology were largely abandoned. The autoimmune hypothesis still remains but has yet to be either been proven nor disproven conclusively.

Original language	English (US)
Title of host publication	Bladder Pain Syndrome - An Evolution
Publisher	Springer International Publishing
Pages	33-36
Number of pages	4
ISBN (Electronic)	9783319614496
ISBN (Print)	9783319614489
DOIs	https://doi.org/10.1007/978-3-319-61449-6_6
State	Published - Jan 1 2017

ASJC Scopus subject areas

General Medicine

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title = "Interstitial cystitis: Animal models",

abstract = "In 1990, although the etiology and pathogenesis of the condition then referred to as “interstitial cystitis” (IC) was admitted to be largely or even completely unknown, the widely held implicit working assumptions held that it is a bladder disease. These assumptions implied that if we could discover and correct what was wrong with the bladder itself then we could cure the condition. These assumptions have proved to be largely off base. At that time, the previous suggestions that IC might have a primarily autoimmune or infectious etiology were largely abandoned. The autoimmune hypothesis still remains but has yet to be either been proven nor disproven conclusively.",

author = "Tony Buffington and Ruggieri, {Michael R.} and Klumpp, {David J.}",

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N2 - In 1990, although the etiology and pathogenesis of the condition then referred to as “interstitial cystitis” (IC) was admitted to be largely or even completely unknown, the widely held implicit working assumptions held that it is a bladder disease. These assumptions implied that if we could discover and correct what was wrong with the bladder itself then we could cure the condition. These assumptions have proved to be largely off base. At that time, the previous suggestions that IC might have a primarily autoimmune or infectious etiology were largely abandoned. The autoimmune hypothesis still remains but has yet to be either been proven nor disproven conclusively.

AB - In 1990, although the etiology and pathogenesis of the condition then referred to as “interstitial cystitis” (IC) was admitted to be largely or even completely unknown, the widely held implicit working assumptions held that it is a bladder disease. These assumptions implied that if we could discover and correct what was wrong with the bladder itself then we could cure the condition. These assumptions have proved to be largely off base. At that time, the previous suggestions that IC might have a primarily autoimmune or infectious etiology were largely abandoned. The autoimmune hypothesis still remains but has yet to be either been proven nor disproven conclusively.

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Interstitial cystitis: Animal models

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