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IVGG therapy in Kawasaki disease: Mechanism(s) of action
Stanford T. Shulman
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Corresponding author for this work
Pediatrics
Research output
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Contribution to journal
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Article
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peer-review
43
Scopus citations
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Keyphrases
Mechanism of Action
100%
Disease Mechanisms
100%
Kawasaki Disease
100%
Etiology
33%
United States
16%
Thrombosis
16%
Echocardiography
16%
Possible Mechanisms
16%
Immune Cells
16%
Japan
16%
Stenosis
16%
Dramatic Effect
16%
Immune Activation
16%
Immunomodulatory Effect
16%
Aneurysm
16%
Myocardial Ischemia
16%
Epidemiologic
16%
Developing World
16%
Cytokine Production
16%
Suppressor T Cells
16%
Etiological Agents
16%
Coronary Arteritis
16%
Anti-inflammatory Effect
16%
Infarction
16%
Acquired Heart Disease
16%
Angiography
16%
Pediatric Diseases
16%
Receptor Blockade
16%
Infectious Etiology
16%
Fc Receptor
16%
Coronary Abnormality
16%
Coronary Artery Abnormalities
16%
Mucocutaneous Lymph Node Syndrome
16%
Neutralization Degree
16%
Antitoxic Effect
16%
Ectasia
16%
Anti-idiotypic Antibody
16%
Medicine and Dentistry
Kawasaki Disease
100%
Diseases
28%
Echocardiography
14%
Immunity
14%
Receptor Blocking
14%
Toxicity
14%
Stenosis
14%
Thrombosis
14%
Infarction
14%
Coronary Artery
14%
Angiography
14%
Cytokine Production
14%
down Regulation
14%
Heart Muscle Ischemia
14%
Clinical Finding
14%
Neutralization
14%
Aneurysm
14%
Antiinflammatory Activity
14%
Fc Receptor
14%
Heart Disease
14%
Immunocompetent Cell
14%
Arteritis
14%
Ectasia
14%
Antiidiotypic Antibody
14%
Pediatrics
14%
Regulatory T Cell
14%
Pharmacology, Toxicology and Pharmaceutical Science
Mucocutaneous Lymph Node Syndrome
100%
Disease
28%
Antiinflammatory Activity
14%
Cytokine
14%
Heart Disease
14%
Toxicity
14%
Infarction
14%
Heart Muscle Ischemia
14%
Fc Receptor
14%
Antiidiotypic Antibody
14%
Thrombosis
14%
Aneurysm
14%
Stenosis
14%
Arteritis
14%