Manganese exposure is cytotoxic and alters dopaminergic and GABAergic neurons within the basal ganglia

Neuroscience

• Basal Ganglia 100%
• Parkinson's Disease 100%
• GABA Receptor 100%
• Dopaminergic 100%
• Tyrosine Hydroxylase 100%
• Substantia Nigra Pars Compacta 100%
• In Vivo 50%
• Striatum 50%
• Cytoskeleton 50%
• Nissl Body 50%
• Dopaminergic Neuron 50%
• In Vitro 50%
• Amino Acid 50%
• Promoter Region 50%
• Acid Protein 50%
• Glutamate Decarboxylase 67 50%
• Red Fluorescent Protein 50%
• Metabolic Pathway 50%
• Extrapyramidal Symptoms 50%

Keyphrases

• Basal Ganglia 100%
• GABAergic Neurons 100%
• Dopaminergic Neurons 100%
• Manganese Exposure 100%
• Manganese 80%
• Parkinson's Disease 40%
• Substantia Nigra Pars Compacta 40%
• Manganese Chloride 40%
• Cell number 20%
• Environmental Exposure 20%
• Vulnerability 20%
• Circuitry 20%
• Striatum 20%
• Neurochemicals 20%
• In Vitro Model 20%
• Essential nutrients 20%
• Amino Acid Metabolism 20%
• Extrapyramidal Symptoms 20%
• Glutamic Acid Decarboxylase 65 (GAD65) 20%
• Overlapping Features 20%
• Tyrosine Hydroxylase Expression 20%
• Red Fluorescent Protein 20%
• Mesencephalic Culture 20%
• Tyrosine Hydroxylase-positive Neurons 20%
• Nissl Body 20%
• Manganese Toxicity 20%
• Neuronal Cytoskeleton 20%
• Amino Lipid 20%

Pharmacology, Toxicology and Pharmaceutical Science

• Dopamine Receptor Stimulating Agent 100%
• 4 Aminobutyric Acid Receptor 100%
• Tyrosine 3 Monooxygenase 66%
• Parkinson's Disease 66%
• Extrapyramidal Symptoms 33%
• Amino Acid 33%
• Environmental Exposure 33%
• Acid Protein 33%
• Glutamate Decarboxylase 67 33%
• Red Fluorescent Protein 33%
• Manganese Chloride 33%