Mice deficient in BACE1, the Alzheimer's β-secretase, have normal phenotype and abolished β-amyloid generation

Yi Luo, Brad Bolon, Steve Kahn, Brian D. Bennett, Safura Babu-Khan, Paul Denis, Wei Fan, Hue Kha, Jianhua Zhang, Yunhua Gong, Laura Martin, Jean Claude Louis, Qiao Yan, William G. Richards, Martin Citron, Robert Vassar*

*Corresponding author for this work

Research output: Contribution to journalArticle

886 Scopus citations
Original languageEnglish (US)
Pages (from-to)231-232
Number of pages2
JournalNature neuroscience
Volume4
Issue number3
DOIs
StatePublished - 2001

ASJC Scopus subject areas

  • Neuroscience(all)

Cite this

Luo, Y., Bolon, B., Kahn, S., Bennett, B. D., Babu-Khan, S., Denis, P., Fan, W., Kha, H., Zhang, J., Gong, Y., Martin, L., Louis, J. C., Yan, Q., Richards, W. G., Citron, M., & Vassar, R. (2001). Mice deficient in BACE1, the Alzheimer's β-secretase, have normal phenotype and abolished β-amyloid generation. Nature neuroscience, 4(3), 231-232. https://doi.org/10.1038/85059