MIR-1343 attenuates pathways of fibrosis by targeting the TGF-ß receptors

Keyphrases

• Transforming Growth Factor-β 100%
• Fibrosis 100%
• Transforming Growth Factor Receptor 100%
• MicroRNA 66%
• Smad3 33%
• Lung Fibrosis 33%
• Chronic Obstructive Pulmonary Disease 33%
• Lung Epithelial Cells 33%
• Airway Inflammation 33%
• Idiopathic Pulmonary Fibrosis 33%
• Cell Migration 33%
• Disease Severity 33%
• Response to Stress 33%
• Therapeutic Use 33%
• Cystic Fibrosis 33%
• 3′-untranslated Region (3′-UTR) 33%
• Respiratory Disease 33%
• Reduced Cell 33%
• Effector Molecules 33%
• Receptor 2 33%
• Epithelial-to-mesenchymal Transition 33%
• Fibrotic Markers 33%
• Fibrotic Lung Disease 33%
• Smad2 33%
• Airway Injury 33%
• A549 33%
• Receptor 1 33%
• Chronic Respiratory Diseases 33%
• Factor Exposure 33%
• Human Neutrophils 33%
• Airway Fibrosis 33%
• Scratch Wound 33%
• Respiratory Obstruction 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Growth Factor Receptor 100%
• Cancer Growth Factor 100%
• Fibrosis 100%
• microRNA 40%
• Chronic Obstructive Lung Disease 20%
• Cytokine 20%
• Lung Fibrosis 20%
• Lung Disease 20%
• Fibrosing Alveolitis 20%
• Wound 20%
• Cystic Fibrosis 20%
• Disease Severity 20%
• Respiratory Tract Disease 20%
• Chronic Respiratory Tract Disease 20%
• Inflammation 20%
• Airway Obstruction 20%

Immunology and Microbiology

• Cancer Growth Factor 100%
• Epithelial Mesenchymal Transition 20%
• Cytokine 20%
• Cell Migration 20%
• Neutrophil 20%
• Airway 20%