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Mitochondria control acute and chronic responses to hypoxia
G. S. McElroy,
N. S. Chandel
*
*
Corresponding author for this work
Medicine, Pulmonary Division
Research output
:
Contribution to journal
›
Article
›
peer-review
76
Scopus citations
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Keyphrases
Acute Changes
33%
Acute Hypoxia
33%
Acute Response
100%
Angiogenesis
33%
Carotid Body
33%
Cell Proliferation
33%
Cell Signaling
33%
Chemosensory Cells
33%
Chronic Hypoxia
33%
Chronic Response
100%
Erythropoietin
33%
Homeostatic Mechanisms
33%
Hypoxia
33%
Hypoxia Response
100%
Hypoxia-inducible Factor
33%
Mitochondria
100%
Mitochondrium
100%
Oxygen Consumption
33%
Pulmonary Arterial Tone
33%
Reactive Oxygen Metabolites
33%
Reactive Oxygen Species Production
33%
Red Blood Cells
33%
Respiration
33%
Smooth muscle Contraction
33%
Vascular Endothelial Growth Factor
33%
Medicine and Dentistry
Angiogenesis
33%
Artery Tone
33%
Carotid Body
33%
Cell Proliferation
33%
Erythrocyte
33%
Erythropoietin
33%
Homeostasis
33%
Hypoxia
100%
Hypoxia Inducible Factor
33%
Metabolite
33%
Mitochondrion
100%
Muscle Contraction
33%
Muscle Fiber Contraction
33%
Oxygen Consumption
33%
Reactive Oxygen Species
33%
Signal Transduction
33%
Vasculotropin
33%
Immunology and Microbiology
Angiogenesis
33%
Artery Tone
33%
Carotid Body
33%
Erythropoietin
33%
Hemocyte
33%
Homeostasis
33%
Hypoxia Inducible Factor
33%
Metabolite
33%
Mitochondrion
100%
Muscle Contraction
33%
Muscle Fiber Contraction
33%
Oxygen Consumption
33%
Signal Transduction
33%
Vasculotropin
33%
Biochemistry, Genetics and Molecular Biology
Angiogenesis
33%
Artery Tone
33%
Erythropoietin
33%
Hemocyte
33%
Homeostasis
33%
Hypoxia-Inducible Factors
33%
Metabolite
33%
Mitochondrion
100%
Muscle Contraction
33%
Muscle Fiber Contraction
33%
Oxygen Consumption
33%
Signal Transduction
33%
Vascular Endothelial Growth Factor
33%