Abstract
In response to growth signals, mechanistic target of rapamycin complex 1 (mTORC1) stimulates anabolic processes underlying cell growth. We found that mTORC1 increases metabolic flux through the de novo purine synthesis pathway in various mouse and human cells, thereby influencing the nucleotide pool available for nucleic acid synthesis. mTORC1 had transcriptional effects on multiple enzymes contributing to purine synthesis, with expression of the mitochondrial tetrahydrofolate (mTHF) cycle enzyme methylenetetrahydrofolate dehydrogenase 2 (MTHFD2) being closely associated with mTORC1 signaling in both normal and cancer cells. MTHFD2 expression and purine synthesis were stimulated by activating transcription factor 4 (ATF4), which was activated by mTORC1 independent of its canonical induction downstream of eukaryotic initiation factor 2α eIF2α phosphorylation. Thus, mTORC1 stimulates the mTHF cycle, which contributes one-carbon units to enhance production of purine nucleotides in response to growth signals.
Original language | English (US) |
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Pages (from-to) | 728-733 |
Number of pages | 6 |
Journal | Science |
Volume | 351 |
Issue number | 6274 |
DOIs | |
State | Published - Feb 12 2016 |
Funding
We thank D. J. Kwiatkowski and R. J. Kaufman for materials and M. Yuan, S. Breitkopf, J. J. Howell, M. Turner, and Y. Zhang for technical assistance. This research was supported by grants from the Bettencourt Foundation and LAM Foundation (I.B.-S.), TS Alliance (G.H.), NSF fellowship DGE-1144152 (S.J.H.R.), and NIH grants K99-CA194192 (I.B.-S.), P01-CA120964 (J.M.A. and B.D.M.), P30-CA006516 (J.M.A.), and R01-CA181390 and R35-CA197459 (B.D.M.). I.B.-S. and G.H. conceived, performed, and analyzed all experiments and prepared the manuscript. S.J.H.R performed the Cancer Genome Atlas analysis. J.M.A. performed LC-MS/MS experiments. B.D.M. directed research, reviewed all data, and prepared the manuscript. All authors reviewed the manuscript and declare no competing financial interests.
ASJC Scopus subject areas
- General