NLRP3 plays a critical role in the development of experimental autoimmune encephalomyelitis by mediating Th1 and Th17 responses

Keyphrases

• Th1 Response 100%
• NOD-like Receptor Protein 3 (NLRP3) 100%
• Experimental Autoimmune Encephalomyelitis 100%
• Th17 Response 100%
• Interleukin-18 (IL-18) 40%
• Multiple Sclerosis 30%
• Spinal Cord 30%
• Innate Immunity 20%
• Histology 10%
• Disease Course 10%
• Mouse Model 10%
• Caspase-1 10%
• Host Response 10%
• Protein Family 10%
• Macrophages 10%
• Interleukin-1β 10%
• Dendritic Cells 10%
• Autoimmune Disease 10%
• Adaptive Immunity 10%
• CD8+ T Cells 10%
• Microglia 10%
• Disease Severity 10%
• Myelin 10%
• Through Effect 10%
• Inflammatory Infiltrate 10%
• CD4+ T Cells 10%
• T Helper 17 (Th17) 10%
• Danger-associated Molecular Patterns 10%
• Pathogen-associated Molecular Patterns 10%
• Leucine-rich Repeat 10%
• Interleukin-17 (IL-17) 10%
• Astrogliosis 10%
• Inflammasome Complex 10%
• Nucleotide-binding Domain 10%
• Cryopyrin 10%
• Pivotal Regulator 10%
• NALP3 10%
• Pyrin Domain Containing 3 10%
• NLRP3 Protein 10%

Immunology and Microbiology

• Experimental Autoimmune Encephalomyelitis 100%
• Multiple Sclerosis 60%
• Spinal Cord 60%
• Interleukin 18 40%
• Adaptive Immune System 20%
• Innate Immune System 20%
• Pathogen-Associated Molecular Pattern 20%
• Dendritic Cell 20%
• Cytotoxic T-Cell 20%
• T-Helper Cell 20%
• Macrophage 20%
• Interleukin-1 20%
• Immune Response 20%
• Protein Family 20%
• Mouse Model 20%
• Autoimmune Disease 20%
• NALP3 20%
• Caspase 1 20%
• Cytokine 20%
• Myelin 20%
• Pyrin Domain 20%
• Interleukin 17 20%
• Inflammasome 20%
• Leucine 20%