INIS
nad
100%
mitochondria
100%
proteins
72%
acetylation
36%
humans
18%
therapy
18%
balances
18%
hypertrophy
18%
heart failure
18%
mice
9%
data
9%
genetics
9%
levels
9%
interactions
9%
energy
9%
mutagenesis
9%
pools
9%
oxidation
9%
transport
9%
increasing
9%
calcium
9%
permeability
9%
precursor
9%
lysine
9%
shuttles
9%
uptake
9%
elevation
9%
adenosine triphosphate
9%
nicotinamide-adenine dinucleotide
9%
Medicine and Dentistry
Heart
54%
Reduced Nicotinamide Adenine Dinucleotide
45%
Mitochondrial Protein
36%
Protein
27%
Heart Failure
18%
Combination Therapy
18%
Acetylation
18%
Protein Acetylation
18%
Development
9%
Mitochondrion
9%
Adenosine Triphosphate
9%
Precursor
9%
Aspartic Acid
9%
Lysine
9%
Calcium Transport
9%
Heart Function
9%
Ventricular Hypertrophy
9%
Mutagenesis
9%
Therapy Effect
9%
Oligomycin
9%
Synthetase
9%
Nicotinamide Adenine Dinucleotide
9%
Malic Acid
9%
Cyclophilin D
9%
Mitochondrial Permeability Transition Pore
9%
Uptake Assay
9%
Analysis
9%
Human
9%
Biochemistry, Genetics and Molecular Biology
Mitochondrial Protein
36%
Protein
27%
Acetylation
18%
Protein Acetylation
18%
Development
9%
Electric Potential
9%
Mutagenesis
9%
Precursor
9%
Synthase
9%
Alpha Oxidation
9%
Mitochondrion
9%
Lysine
9%
Heart Function
9%
Calcium Transport
9%
Adenosine Triphosphate
9%
Human
9%
Energy
9%
Nicotinamide Adenine Dinucleotide
9%
Proteome
9%
Genetic Model
9%
Mouse
9%
Mitochondrial Permeability Transition Pore
9%