Novel tissue level effects of the Staphylococcus aureus enterotoxin gene cluster are essential for infective endocarditis

Keyphrases

• Adjunct Therapy 10%
• Cluster Deletion 10%
• Deletion Strain 10%
• Disease Causation 10%
• Endothelial Barrier 10%
• Enterotoxin Gene Cluster 100%
• Epithelial Barrier 10%
• Human Intravenous Immunoglobulin 10%
• Illness 10%
• Immune Activation 10%
• Infective Endocarditis 100%
• Intravenous Immunoglobulin (IVIg) 20%
• Lethality 10%
• Level Effect 100%
• Life-limiting Conditions 10%
• Local Effects 20%
• Mucosal Surfaces 10%
• Rabbit Model 10%
• S. Aureus Infection 10%
• Sepsis 70%
• Staphylococcal Enterotoxins 100%
• Staphylococcus Aureus 20%
• Study Support 10%
• Superantigen 60%
• Tissue Effects 10%
• Tissue Level 100%
• Toxic Shock Syndrome 10%
• Toxic Shock Syndrome toxin-1 (TSST-1) 10%
• Vancomycin 20%
• Vegetation Formations 10%
• Virulence Factors 10%

Pharmacology, Toxicology and Pharmaceutical Science

• Bacterial Endocarditis 100%
• Cerebrovascular Accident 10%
• Diseases 30%
• Enterotoxin 100%
• Immunoglobulin 30%
• Infection 10%
• Lethality 10%
• Rabbit Model 10%
• Sepsis 70%
• Staphylococcus Aureus 100%
• Superantigen 60%
• Toxic Shock Syndrome 10%
• Toxic Shock Syndrome Toxin 1 10%
• Vancomycin 20%
• Virulence Factor 10%

Neuroscience

• Adjuvant Therapy 10%
• Cell Activation 10%
• Cerebrovascular Accident 10%
• Gene Cluster 100%
• In Vitro 10%
• Infective Endocarditis 100%
• Intravenous Immunoglobulin 30%
• Superantigen 60%
• Vancomycin 20%