Oppositely imprinted genes p57(Kip2) and Igf2 interact in a mouse model for Beckwith-Wiedemann syndrome

Keyphrases

• Mouse Model 100%
• Insulin-like Growth Factor 2 (IGF2) 100%
• Beckwith-Wiedemann Syndrome 100%
• Imprinted Genes 100%
• P57Kip2 100%
• Macroglossia 33%
• Growth Factors 16%
• Null mutation 16%
• Deregulation 16%
• Gain-of-function mutation 16%
• Large-scale Cluster 16%
• Increased Vulnerability 16%
• Childhood Brain Tumors 16%
• Expressed Genes 16%
• Cyclin-dependent Kinase Inhibitor 16%
• Dysplasia 16%
• Loss of Imprinting 16%
• Human Chromosomes 16%
• Specific Growth 16%
• Cleft Palate 16%
• Chromosome 11p15 16%
• Omphalocele 16%
• Abdominal Wall Defect 16%
• G1 Cyclins 16%
• Polydactyly 16%
• Somatic Overgrowth 16%
• Visceromegaly 16%
• Kidney Dysplasia 16%
• Placentomegaly 16%

Biochemistry, Genetics and Molecular Biology

• Mouse Model 100%
• Beckwith-Wiedemann Syndrome 100%
• Dysplasia 33%
• Loss of Function Mutation 16%
• Human Chromosome 16%
• Growth Factor 16%
• Cyclin-Dependent Kinase 16%