Proteolytic N-terminal truncation of cardiac troponin I enhances ventricular diastolic function

Biochemistry, Genetics and Molecular Biology

• Troponin I 100%
• Cardiac Troponin 90%
• N-Terminus 50%
• Heart Left Ventricle Enddiastolic Pressure 20%
• Phosphorylation 10%
• Transgenic Mouse 10%
• Adrenergic Stimulation 10%
• Protein Catabolism 10%
• Wild Type Mouse 10%
• Protein Kinase A 10%
• Isoform 10%
• Heart Function 10%
• ATPase 10%
• Hemodynamic 10%
• Heart Output 10%
• Myofibril 10%
• Adaptation 10%
• Heart Preload 10%
• Experiment 10%
• Regulatory Mechanism 10%
• Protein Hydrolysis 10%

INIS

• control 30%
• relaxation 30%
• comparative evaluations 20%
• adrenergics 20%
• mice 10%
• levels 10%
• in vivo 10%
• proteins 10%
• transgenic mice 10%
• stimulation 10%
• regulations 10%
• muscles 10%
• kinases 10%
• phosphorylation 10%
• increasing 10%
• affinity 10%
• cleavage 10%
• removal 10%
• proteolysis 10%
• cardiac output 10%