Abstract
Like other herpesviruses, Epstein-Barr virus persists in its host through its ability to establish a latent infection that periodically reactivates. Latent membrane protein 2A (LMP2A) regulates reactivation from latency by interfering with normal B cell signal transduction processes, and may define a new class of regulators of herpesvirus latency.
Original language | English (US) |
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Pages (from-to) | 39-42 |
Number of pages | 4 |
Journal | Trends in Microbiology |
Volume | 4 |
Issue number | 1 |
DOIs | |
State | Published - Jan 1996 |
ASJC Scopus subject areas
- Microbiology (medical)
- Infectious Diseases
- Virology
- Microbiology