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Signaling Pathways in Endometriosis (Eutopic/Ectopic)
J. Julie Kim
*
, Xunqin Yin
*
Corresponding author for this work
Obstetrics and Gynecology
Research output
:
Chapter in Book/Report/Conference proceeding
›
Chapter
5
Scopus citations
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Medicine and Dentistry
Cyclic AMP Dependent Protein Kinase
25%
Cytokine
25%
Diseases
25%
Endometriosis
75%
Gene Expression
50%
Lesion
25%
Mammalian Cell
50%
Mediator
25%
Mitogen Activated Protein Kinase Kinase
25%
Protein
25%
Protein Kinase
25%
Second Messenger
25%
Signal Transduction
75%
Signal Transduction Pathway
50%
Signaling Mechanism
75%
Stimulus
25%
Survival
25%
Tissue Cell
25%
INIS
cytokines
25%
defects
25%
diseases
25%
genes
50%
information
25%
kinases
75%
mitogens
25%
proliferation
75%
proteins
100%
signals
25%
stimuli
25%
tissues
25%
yeasts
25%
Biochemistry, Genetics and Molecular Biology
Cytokine
25%
Gene Expression
50%
Mediator
25%
Mitogen-Activated Protein Kinase Kinase
25%
Protein Kinase
25%
Protein Kinase A
25%
Second Messenger System
25%
Signal Transduction
75%
Survival
25%
Yeast
25%
Neuroscience
Cell Signaling
75%
Gene Expression
50%
Mitogen-Activated Protein Kinase Kinase
25%
Protein
25%
Protein Kinase A
25%
Protein Kinases
25%
Second Messenger System
25%