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The pathogenetic role of apoptosis in hypercoagulable states
J. Wang
*
,
H. C. Kwaan
*
Corresponding author for this work
Medicine, Hematology Oncology Division
Preventive Medicine
Research output
:
Contribution to journal
›
Article
›
peer-review
1
Scopus citations
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Medicine and Dentistry
Programmed Cell Death
100%
Procoagulant
100%
Diseases
50%
Pathogenesis
50%
Cell Type
50%
Endothelial Cell
50%
Blood Clotting
50%
Anticoagulation
50%
Cytokine
25%
Tumor Cell
25%
Hemostat
25%
Thromboplastin
25%
Monocyte
25%
Phosphatidylserine
25%
Thrombosis
25%
Cardiovascular System
25%
Cancer Procoagulant
25%
Vascular Smooth Muscle Cell
25%
Coagulation
25%
Vascular Endothelial Cell
25%
Hypercoagulability
25%
Bacterium Lipopolysaccharide
25%
Thrombomodulin
25%
Cells
25%
Macrophage
25%
Atherosclerosis
25%
INIS
apoptosis
100%
pathogenesis
50%
anticoagulants
50%
cancer
25%
levels
25%
tissues
25%
correlations
25%
tumor cells
25%
risks
25%
balances
25%
diseases
25%
reviews
25%
inflammation
25%
cytokines
25%
macrophages
25%
muscles
25%
monocytes
25%
lipopolysaccharides
25%
thrombosis
25%
atherosclerosis
25%
hemostatics
25%