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The Tubulointerstitial Pathophysiology of Progressive Kidney Disease
H. William Schnaper
*
*
Corresponding author for this work
Pediatrics
Research output
:
Contribution to journal
›
Review article
›
peer-review
109
Scopus citations
Overview
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Keyphrases
Progressive Kidney Disease
100%
Pathophysiology
100%
Inflammation
100%
Fibrogenic
100%
Nephron
100%
Healing
100%
Renal Tubule
100%
Renal Tubular Epithelial Cells
100%
Hypoxia
100%
Myofibroblast
100%
Tubulointerstitial
100%
Endoplasmic Reticulum Stress (ER stress)
100%
CKD Progression
100%
Downward Spiral
100%
Acidosis
100%
Tissue Loss
100%
Vicious Cycle
100%
Generating Capacity
100%
One-phase
100%
Tubular Transport
100%
Anaerobic Metabolism
100%
Nephron Loss
100%
Maladaptive Responses
100%
Nephron Hypertrophy
100%
Medicine and Dentistry
Pathophysiology
100%
Nephropathy
100%
Nephron
100%
Hypoxia
33%
Epithelial Cell
33%
Fibrogenesis
33%
Myofibroblast
33%
Endoplasmic Reticulum Stress
33%
Metabolic Acidosis
33%
Anaerobic Metabolism
33%