The tyrosine hydroxylase-human growth hormone (GH) transgenic mouse as a model of hypothalamic GH deficiency: Growth retardation is the result of a selective reduction in somatotrope numbers despite normal somatotrope function

Rhonda D. Kineman, Grazia Aleppo, Lawrence A. Frohman*

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

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Biochemistry, Genetics and Molecular Biology

Pharmacology, Toxicology and Pharmaceutical Science

Neuroscience