Versican V1 Proteolysis in Human Aorta in Vivo Occurs at the Glu 441-Ala442 Bond, a Site That Is Cleaved by Recombinant ADAMTS-1 and ADAMTS-4

John D. Sandy*, Jennifer Westling, Richard D. Kenagy, M. Luisa Iruela-Arispe, Christie Verscharen, Juan Carlos Rodriguez-Mazaneque, Dieter R. Zimmermann, Joan M. Lemire, Jens W. Fischer, Thomas N. Wight, Alexander W. Clowes

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

392 Scopus citations

Abstract

Mature human aorta contains a 70-kDa versican fragment, which reacts with a neoepitope antiserum to the C-terminal peptide sequence DPEAAE. This protein therefore appears to represent the G1 domain of versican VI (G1-DPEAAE 441), which has been generated in vivo by proteolytic cleavage at the Glu441-Ala442 bond, within the sequence DPEAAE 441-A442RRGQ. Because the equivalent aggrecan product (G1-NITEGE341) and brevican product (G1-EAVESE395) are generated by ADAMTS-mediated cleavage of the respective proteoglycans, we tested the capacity of recombinant ADAMTS-1 and ADAMTS-4 to cleave versican at Glu441-Ala442. Both enzymes cleaved a recombinant versican substrate and native human versican at the Glu441-Ala 442 bond and the mature form of ADAMTS-4 was detected by Western analysis of extracts of aortic intima. We conclude that versican VI proteolysis in vivo can be catalyzed by one or more members of the ADAMTS family of metalloproteinases.

Original languageEnglish (US)
Pages (from-to)13372-13378
Number of pages7
JournalJournal of Biological Chemistry
Volume276
Issue number16
DOIs
StatePublished - Apr 20 2001

ASJC Scopus subject areas

  • Molecular Biology
  • Biochemistry
  • Cell Biology

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