XA histone modifier's ill-gotten copy gains

Ryan Rickels, Ali Shilatifard*

*Corresponding author for this work

Research output: Contribution to journalShort surveypeer-review

2 Scopus citations

Abstract

Little is known about the molecular machinery that contributes to site-specific copy number variations or how CNVs fit into the chronology of tumor progression. Black et al. (2013) now demonstrate that the overexpression of a histone demethylase induces transient copy gain of specific genomic loci known to harbor proto-oncogenes.

Original languageEnglish (US)
Pages (from-to)X477-479
JournalCell
Volume154
Issue number3
DOIs
StatePublished - Aug 1 2013

ASJC Scopus subject areas

  • Biochemistry, Genetics and Molecular Biology(all)

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